Thanks a lot for ur help mate,
first of all, I checked my body fat and Im at 18%….
so as u said, it is either bulk or cut , Ive done a cycle for me and I want ur advise, (last one 🙂 )
week 1-4 test pro 150mg eod( mon-wed-fri)
week 1-10 test enan 350mg twice a week
week 11-12 test pro 150 eod( mon-wed-fri)
week 1-12 arimidex eod
week 1-6 dbol 30mg ed
week 13-14 rest
week 15-19 pct nolvadex.
test e and p are from concent rex.. called them enanTREX and propiTREX. (legit)
I want to know if this cycle sounds good?? and some help with the PCT please. and of course Im prepared to make changes…..
hope to hear from u soon, Im keen to start ASAP. and again thanks a lot mate.
Synovial biopsy specimens from 20 patients with rheumatoid arthritis were subjected to quantitative analysis for several parameters of inflammation and for enumeration of synovial tissue mast cells. Strong positive correlations were found between numbers of mast cells per cubic millimeter of synovial tissue and the following synovial tissue parameters: inflammatory index (a quantification of lymphocytic infiltration), Leu-3a grade (T helper/inducer lymphocytes), Leu-1 grade (T lymphocyte), and plasma cell grade. A strong negative correlation was found between the synovial mast cell count and the extent of sublining layer fibrin deposition. Correlations between synovial mast cell count and Leu-2a grade, ratio of Leu-3a grade:Leu-2a grade, OKM1 grade, HLA-DR grade, and lining layer thickness grade did not reach statistical significance. In addition, we obtained synovial specimens from 6 of the patients both before and after long-term therapy with oral methotrexate and from 3 of the patients before, and 1 week after, an intraarticular injection of steroid. The 3 patients who had an intraarticular steroid injection showed a 67-96% decrease in the number of synovial tissue mast cells; there was no significant change in the number of synovial mast cells in the tissues of the 6 patients who received oral methotrexate. These observations are the first documentation of a quantitative relationship between the number of mast cells and the number and phenotypic profile of infiltrating lymphocytes in an inflamed tissue, which in this case, is human synovium. Our findings suggest that mast cells are involved in the pathologic interactions in rheumatoid arthritis and might play a role in the early phases of exacerbations of disease activity.